Chapter 8

Notes (19 August 2008) 

P. 244: If the transmembrane A.C. voltage of the OHC is in fact capacitance rather than resistance coupled, as in the model of Dallos and Evans (1995a, b; Fig. 5.21A), how could changing the membrane resistance (e.g. by opening the SK2 channels) alter the magnitude of the motile process (via altering the A.C. transmembrane voltage, assuming that it is this that drives the motility)? (19 August 2008)

P. 249: Why does the MOC fast effect not also protect from trauma, if it reduces the amplitude of the travelling wave? It is not clear. In fact, the degree of active amplification of the travelling wave at high stimulus intensities, where the OHC are saturated, is uncertain. (19 August 2008)

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